Prof. Rohit Saxena

Dr R P Centre for Ophthalmic Sciences, AIIMS, New Delhi.

1 article

Prof. Rohit Saxena is Head of Strabismus and Neuro Ophthalmology Services at the Dr Rajendra Prasad Centre for Ophthalmic Sciences, All India Institute of Medical Sciences, New Delhi. He completed his medical and specialty training MBBS, MD (Ophthalmology) and PhD from the All India Institute of Medical Sciences. He has numerous awards to his credit including the Best of Show Video at the American Academy of Ophthalmology 2019, Best Video SPOSI Annual Conference 2019, AIIMS Excellence Award 2018, Fan Favorite Video at the AAO 2016 and the AAO and APAO Achievement awards. He has 200 indexed publications and has authored over 60 chapters in national and international books. He is regularly conducting Instruction course at AIOS and is a co-instructor of numerous ICs in Squint and Neuro-Ophthalmology for past 15 years. He is an Invited speaker at numerous National Conferences and CME meets and has also performed live squint surgery in national and state level conferences. He is the principal investigator of numerous multicentric clinical trials and is involved in numerous research projects.

  1. Articles 28 Jul 2021 16 min read

    Papilledema- A Ready Reckoner for Postgraduates

    Introduction Papilledema is used to describe optic disc edema resulting from increased intracranial pressure while disc edema could be due to any cause. While disc edema can be unilateral or bilateral, papilledema is usually bilateral, however, it can be asymmetric, to begin with. Another important differential of true papilledema is pseudo papilledema which is seen in conditions like a small hyperopic optic disc or optic disc drusen, etc. This chapter shall entail a discussion on the etiopathogenesis of papilledema and the approach to diagnosis and management of papilledema. Papilledema and its mechanism It’s a form of bilateral optic disc swelling associated with increased intracranial pressure. An increase in intracranial pressure can occur secondary to an intracranial space-occupying lesion, cerebral venous thrombosis, or can be idiopathic as in the case of Idiopathic Intracranial hypertension (IIH). Figure 1 depicts the mechanism of the development of papilledema. Pseudo vs true p