Dr. Hema Joshi

MBBS, DO, DNB

Consultant, Cataract, Cornea & Ocular surface, Director, Academics, Gomabai Netralaya, Neemuch

6 articles

Dr. Hema Joshi completed her MBBS from Netaji Subhash Chandra Bose Medical College, Jabalpur. She completed her Diploma in Ophthalmology from Mahatma Gandhi Memorial Medical College and Maharaja Yashwant Rao Hospital, Indore and DNB from Medical Research Foundation, Sankara Nethralaya, Chennai. She spent some time in the Moorfields Eye Hospital, London, UK as observer. She worked as Consultant, Cataract and General Ophthalmology in Vasan Eye Care, Chennai for 2 years. She underwent fellowship training in Cornea and Ocular surface from PBMAs H V Desai eye Hospital, Pune. She is worked as Consultant, Cataract and Cornea, in H V Desai eye hospital Pune. Currently, she is working as anterior segment consultant and director of academics in Gomabai Netralaya, Neemuch, MP.

  1. Articles 2 Jan 2024 11 min read

    Iris Repair Techniques

    Trauma to the iris can present in various ways depending on its severity and location. Tear at the iris root leads to Iridodialysis, while tears at the pupillary border can damage the sphincter muscle, leading to a partially reactive, atonic or Mydriatic pupil. Tears can be full thickness or partial thickness. Full thickness defect involves the iris stroma and partial thickness involves only the posterior pigment epithelium leading to trans-illumination defects. Traumatic Iridodialysis Traumatic Mydriasis Etiology of iris defects: Iris defects can be a consequence of either: Congenital (iris coloboma) Acquired Iatrogenic (iris lesion removal) Traumatic (traumatic mydriasis or injury resulting in pupil irregularity or muscle tear) Complicated intraocular surgery (can cause dialysis, atonic dilated pupil or iris tissue loss and injury) Symptoms: Patients with photic symptoms due to iris defects complain of: Glare, Haloes in bright light conditions, Diplopia, and Trouble reading Patients

  2. Articles 3 Jan 2022 11 min read

    i-File: Pseudophakic Bullous Keratopathy

    A 60-year-old-male presented to the Cornea clinic with diminution of vision in the left eye for 4 months which was associated with intermittent photophobia and colored haloes around lights, especially on waking up in the morning. The patient also complains of pain and watering. No history of associated redness, itching, or discharge. Past Ocular History: H/o OD cataract surgery 6 years, OS cataract surgery 4 months back. No h/o trauma. Past Medical History/ History of Medication: No significant systemic history could be elicited. The patient was not on any medication. No addictions. Family History: No significant family history found Review of Systems/Systemic Examination: Systemic examination was within normal limits. All vitals were within normal limits. OCULAR EXAMINATION Best Corrected Visual Acuity (Snellen) Right eye (OD): 6/9 Left eye (OS): CF 2 m Ocular Motility/Alignment - Full, free, and painless in all gazes Intraocular Pressure (IOP) OD: 18 mm of Hg OS: 14 mm of Hg Pupils-

  3. Students Gallery 12 Apr 2021 12 min read

    i-File: Fuchs Endothelial Dystrophy

    A 55-year-old female presented to the Cornea clinic with gradually progressive diminution of vision in both eyes for 1 year, which was associated with intermittent photophobia and colored haloes around lights, especially on waking up in the morning. The blurry vision gradually became clearer as the day progressed. No history of associated pain, watering, redness, itching, or discharge. Past Ocular History: No history of ocular trauma or surgery in the past Past Medical History/ History of Medication: No significant systemic history could be elicited. Patient was not on any medication. No addictions. Family History: No significant family history found Review of Systems/Systemic Examination Systemic examination was within normal limits. All vitals were within normal limits. OCULAR EXAMINATION Best Corrected Visual Acuity (Snellen) Right eye (OD): 6/36 Left eye (OS): 6/12 Ocular Motility/Alignment Full, free, and painless in all gazes Intraocular Pressure (IOP) OD: 16 mm of Hg OS: 16 mm o

  4. Articles 1 Apr 2021 20 min read

    Acanthamoeba Keratitis: A review for the Postgraduates

    Eye diseases affecting the cornea are a major cause of blindness worldwide. Among different infectious agents, bacteria, fungi, viruses and protozoans may be causes of keratitis. This article is about the keratitis caused by protozoa. Three amoebic parasites are thought to be significant to human disease, entamoeba which is responsible for amoebic dysenter, naegleria which causes amoebic meningoencephalitis and acanthamoeba which is known for causing keratitis and granulomatous amoebic encephalitis. Acanthamoeba is a ubiquitous, free living protozoa of the subphyla Sarcodina. It is considered as opportunistic pathogen in humans. Acanthamoeba keratitis is a potentially devastating corneal infection which can lead to severe visual loss. Epidemiology: Acanthamoeba keratitis (AK), is rare in the general population (estimated incidence: 1.4 per million person- year) but much frequent in contact lens wearers. The incidence of the disease in developed countries is approximately 1-33 cases per

  5. Articles 1 Apr 2021 16 min read

    Herpetic Eye Disease Study: All You Should Know

    Herpes simplex virus (HSV) keratitis is the leading cause of corneal blindness in developed as well as developing countries. The ocular disease affects approximately 10 million people worldwide. Herpes viruses are a group of double-stranded DNA viruses with three subfamilies: alpha-, beta- and gamma- herpes viruses. HSV-1, HSV-2, and varicella-zoster virus (VZV) belong to the alpha-subfamily. HSV spreads by direct contact with virus shed by saliva or genital secretions. During primary infection, HSV-1 infiltrates the corneal epithelial cells. Here it lyses the host cells and releases virions which in turn infect neighboring cells. Then the HSV travels via neuronal cells to trigeminal ganglion and establishes a latent infection. HSV can remain latent for lifetime by storing its genome in the nucleus of the host neuronal cell. While latent, it produces latency-associated transcripts (LATs) that maintain the integrity of the viral genome. The latent HSV-1 can reactivate and return to the

  6. Articles 1 Apr 2021 40 min read

    Instruments used in Corneal Procedures and Surgeries

    Although tales and myths of ocular surgery and organ transplantation appear in numerous ancient texts, the earliest accounts of ophthalmic diseases were written as a treatise by Sushruta, in the 6th century BC. Sushruta described 76 eye diseases, out of which 51 required surgical treatment. In the western world, the concepts of corneal surgery date back to Greek physician Galen (130-200AD). The concepts to improve corneal clarity by surgery were developed over the next centuries and especially in the 17th and 18th centuries. But the major scientific progress and experimentation in the field of corneal surgeries happened in the 19th century. The progress in corneal surgery was hindered by a lack of understanding of immunology, asepsis, surgical procedures, and of course, the lack of proper instrumentation. Emmert Wolf wrote over a century ago, “A man is only as good as his tools.” Early surgeons were hampered by a lack of proper surgical instruments. Over the centuries, innumerable opht