FVRF (Sankara Nethralaya)

Consultant, Narayana Nethralaya, Bangalore

1 article

He completed his research-cum-clinical Vitreoretina fellowship at Medical Research Foundation, Sankara Nethralaya, Chennai, from 2008-2011. With an experience of 12 years in the field of Vitreoretina, he is currently working as a consultant in Narayana Nethralaya, Bangalore and has a keen interest in academics, research and patient care. His areas of interests include management of retinal vascular disorders like diabetic retinopathy and vascular occlusions, ARMD and minimally invasive vitreous surgery (MIVS) for management of retinal detachments, proliferative diabetic retinopathy, trauma and endophthalmitis. In his previous engagement with Dr. Shroffs Charity Eye Hospital, Delhi, he was in-charge for the screening of preterm babies for retinopathy of prematurity (ROP). To his credit, he has many research publications in national and international peer-reviewed journals. His keen area of research has been retinal imaging in different retinal and choroidal diseases.

  1. Articles 21 May 2023 9 min read

    Seven Important OCT Signs in CNVM

    1. Type of macular neovascularisation (MNV): Macular neovascularisation (MNV) is a broad disease entity characterised by the development of the abnormal vessels arising either from the retinal vasculature (deep capillary plexus) or choroidal vasculature (choriocapillaris). Choroidal neovascular membrane (CNVM) is a subtype of MNV, characterised by the abnormal growths of blood vessels from the choroidal capillaries that penetrate the retina, specifically into the sub-RPE and subretinal spaces. These vessels are extremely immature and extremely fragile, so they bleed and leak, de-structuring the macula, causing severe visual impairment. The classification of MNV is depicted below in Figure 1: Figure 1: A simple classification of macular neovascularisation (MNV): 2. Cause of CNVM: A CNVM is primarily classified based on the etiology into two categories: A) Age-related Macular Degeneration (AMD) associated CNVM and B) Non-AMD associated CNVMs. Causes of non-AMD associated CNVMs: Central s