Dr. Reema Bansal

Additional Professor,Advanced Eye Centre, Post Graduate Institute of Medical Education and Research, Chandigarh, India-160012

3 articles

Dr Reema Bansal MBBS, MS (Ophthalmology), PhD is currently Additional Professor Advanced Eye Centre PGIMER, Chandigarh India. She Has given multiple Invited Guest Lectures in various International and National meetings. She has multiple publications in Scientific Journals 127 (Total), She also authored many Chapters in Books. She has been reviewing editor for Ocular Immunology and Inflammations and assistant editor for (Medical Retina Section) Indian Journal of Ophthalmology. She is presently the reviewer for multiple journals like Ocular Immunology and Inflammation,Journal of Ophthalmic Inflammations and Infections, JAMA Ophthalmology , Eye ,Journal of Ophthalmology, Indian Journal of Ophthalmology. She has received multiple awards like Gold Medal in essay writing competition in the 27th Annual Gujarat Ophthalmology conference at MOUNT ABU 1999. Carl P Herbort award at the National conference of Uveitis Society of India at PGIMER, Chandigarh, 2010. Best poster award at VRSI Conference at Hyderabad 2011. Best E-Poster award EYECON 2012.J M Pahwa Best Paper award XXIst Annual meeting of Vitreoretina society of India, Guwahati 2012. AIOS Rema Mohan Award for Best Paper 72nd AIOC, Agra, 2014. Certificate of Merit for the year 2019 by Indian Journal of Ophthalmology .

  1. Articles 1 Apr 2021 11 min read

    How do we treat inflammatory CNVM ?

    Inflammatory choroidal neovascular membranes (CNVM) cause an acute and significant visual loss 1,2. Mostly affecting eyes with posterior or panuveitis, the chronic recurrent nature of inflammation further worsens the visual prognosis in these eyes, such as serpiginous like choroiditis, punctuate inner choroidopathy (PIC), multifocal choroiditis, Vogt-Koyanagi-Harada (VKH) disease, presumed ocular histoplasmosis syndrome, toxoplasma retinochoroiditis, etc,.3,4 . The angiogenic stimulus in inflammatory CNVM is driven by local inflammation and/or a degenerative breach in retinal pigment epithelium (RPE)-Bruch’s membrane complex4. An active intraocular inflammation induces the release of several inflammatory mediators including cytokines, chemokines, vascular endothelial growth factor (VEGF) and complement activation, which in turn promote retinal and choroidal neovascularization. Majority of inflammatory CNVM are classic (on fundus fluorescein angiography), and type 2 on spectral domain o

    Dr. Krinjeela Bazgain · Dr. Nitin Kumar · +1 other
  2. Articles 1 Apr 2021 13 min read

    Ocular Tuberculosis

    Ocular involvement in TB has been recognized for a long time and was first described in 17111. The spectrum of tuberculosis (TB)-related uveitis is wide. Its diagnosis still remains a challenge due to the lack of uniformity in the diagnostic criteria along with difficulties encountered in confirming the diagnosis by the laboratory methods available. It is a major concern in countries endemic for TB, and intraocular TB is being increasingly reported from various regions2-4. The true prevalence of intraocular TB is not known in India. While it has been reported to be 0.39% in South India, it is much higher (9.86%) in North India1,5. Varying rates of its prevalence have been reported from the world over (0.5% in the U.S.A., 6.31% in Italy, 6.9% in Japan, and 10.5% in Saudi Arabia). The largest series of patients with presumed intraocular TB and their outcome has been recently reported from our center6. Pathogenesis Uveitis due to latent TB has been reported since long. However, their exac

    Dr. Reema Bansal · Prof. Vishali Gupta · +1 other
  3. Articles 5 Apr 2020 15 min read

    Retinal Vasculitis

    Retinal vasculitis is an inflammatory disease of the blood vessels of the retina that may be associated with primary ocular conditions or with inflammatory or infectious diseases in other parts of the body (systemic diseases). It has been defined as the vascular leakage and staining of vessel walls on fluorescein angiography, with or without the clinical appearance of fluffy, white perivascular infiltrates in an eye with evidence of inflammatory cells in the vitreous body or aqueous humor 1. Typically, retinal vasculitis is divided into entities localized to the retina and into systemic diseases involving the eye. These include certain ocular disorders, systemic autoimmune disorders, and some infectious diseases. Etiology: The common primary causes of retinal vasculitis where vessel is the primary target of the inflammatory process are: (a) Localized to the eye - Idiopathic - Intermediate uveitis of the pars planitis type - Frosted branch angiitis - Idiopathic retinal vasculitis, aneur

    Dr. Reema Bansal · Prof. Amod Gupta